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However, Aja and colleagues found no evidence for this hypothesis since in this model CPT-1 should inhibit feeding by increasing cytosolic fatty-acyl CoA levels while they actually showed the initial response of mice to a CPT-1 inhibitor was an increase in appetite [75]
Cardiovascular disease In cardiovascular disease, Myc and Myc-related factor X (Max) upregulate the Gln transporters SLC1A5 and SLC7A5 and mitochondrial malate in pulmonary hypertension, thereby promoting glutaminolysis-induced right ventricular hypertrophy
10.1111/jnc.13544 J
