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Given that cardiomyocyte hypertrophy and interstitial fibrosis are the core pathological features of RV decompensation in PH, it is highly plausible that the dysregulated lactate-lactylation axis acts as a critical, yet underexplored, pathological driver in this progression

15 min before a dose of vehicle (water by oral gavage)

4d), among which six ferroptosis-inhibiting genes (Slc7a11, Slc40a1, Gclm, Fth1, Gclc, and Gss) were upregulated, one ferroptosis-promoting gene (Slc39a14) was downregulated, and one ferroptosis-promoting gene (Hmox1) was upregulated

We recommend: Receiving thorough training from a healthcare professional on proper injection technique

This is likely mediated by activation of the glutamine-inosine-CpxA/R-OmpF pathway 12 (see Fig