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Excess free copper ions can increase reactive oxygen species (ROS) levels through the Fenton reaction, enhancing oxidative stress and cellular damage (140)

Their activation is triggered by the recognition of pathogen-associated molecular patterns (PAMPs), such as bacterial flagellin, and damage-associated molecular patterns (DAMPs), including uric acid crystals

This is a bit of a mouthful, but you talk about the re-localization of chromatin modifiers, RCM hypothesis in your book

This review summarizes the mechanisms by which lipid peroxidation contributes to sarcopenia, with a focus on its regulatory effects on cell membrane damage, mitochondrial dysfunction, and cell death

Suppressing thyroid hormone signaling preserves cone photoreceptors in mouse models of retinal degeneration