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Comparative Analysis: Pharmacological agents like corticosteroids and NSAIDs [56, 110] provide rapid inflammation reduction but risk rupture, unlike TGF- inhibitors [57, 132,133,134] that target fibrosis more precisely, though with variable subtypes [133]

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Structural basis for the retroreduction of inactivated peroxiredoxins by human sulfiredoxin

HCV29RP/MRD cells display metabolomic and proteomic differences that suggest enhancement of mitochondrial activity

Protective loss-of-function mutations in SLC30A8 were first identified through gene burden and population-level analyses [10]