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This intricate nexus, predominantly involving peripherally activated immune cells, notably circulating monocytes, and microglia, along with its repercussions on signaling pathways like serotonin, dopamine, and corticotropin-releasing hormone, with the latters implications on the hypothalamus-pituitaryadrenal axis, initiates sickness behaviors, including fatigue (4, 8)

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This review aims to serve as a comprehensive reference for clinical and laboratory findings for those who are confronted with pediatric and adult patients with a possible diagnosis of a mitochondrial lcFAOD

[DOI] [PMC free article] [PubMed] [Google Scholar] 426.Schafernak KT