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Thyroxine increases renal blood flow and GFR via activation of the renin-angiotensin-aldosterone system (RAAS), increased activity of the sympathetic nervous system and decreased peripheral vascular resistance

however, in the initial stage of atherosclerosis, turbulent blood flow interrupts these connections, making them more permeable, or leaky, and allowing lipoproteins to penetrate into the space under the endothelial cells, known as the intima

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341 Schwann cell injury In DPN, hyperglycemia and oxidative stress lead to Schwann cell dysfunction and apoptosis, resulting in demyelination and impaired nerve conduction