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However, we are increasingly aware that lesser degrees of HTG may result from genetic variants in one of many molecular factors that regulate LPL-mediated lipoprotein metabolism, rather than variations in the LPL gene/protein itself ( The PHP and serum activation/inhibition protocols described here are validated radiometric assays that allow accurate measurement of LPL activity using physiologically relevant lipid substrates, which can be tailored to answer the specific research question at hand

Family History, Actinic Damage and Phenotypic Factors

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