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J.ZhaoF
Studies in mouse models have demonstrated that depletion of C1q results in impaired elimination of redundant synapses, highlighting the role of C1q following stroke [62]
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This way the glutathione enters the bloodstream quickly, providing a continuous antioxidant defence that complements the topical treatment from aox ferulic
Hydroxyflavone metal complexes-molecular structure, antioxidant activity and biological effects
