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This reprograms the tumor microenvironment, restores CTL activity, and enhances antitumor immunity (39)
doi: 10.1016/j.tins.2019.01.003, 85
This study not only validates the feasibility of the mitochondrial dysfunction-induced cell death strategy, but also demonstrates the translational potential of biomimetic drug delivery systems in metabolism-targeted therapy
BPC-157 appears to promote controlled angiogenesis in these environments, not the chaotic vessel formation youd worry about in an oncology context, but organized vascular ingrowth that supports matrix remodeling
Antioxidant support inside brain cells
