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Zhao J, Taverne MA, van der Weijden GC, Bevers MM, van den Hurk R: Immunohistochemical localisation of growth hormone (GH), GH receptor (GHR), insulin-like growth factor I (IGF-I) and type I IGF-I receptor, and gene expression of GH and GHR in rat pre-antral follicles

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Disruption of GSH dynamics typically impairs redox buffering capacity, often manifested as reduced mitochondrial GSH (mtGSH) levels

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Ultimately, the pathological consequences of the immune response associated with ALD include elimination of cell death debris and proliferation of hepatocytes, which may result in complete recovery or unsolved repair manifesting as fibrosis/cirrhosis
