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The resistance to tumor necrosis factor-alpha (TNF)-induced apoptosis of some tumors (Tsujimoto et al., 1985) is attributed to TNF-mediated activation of NFB (Beg and Baltimore, 1996
Increased oxidative damage and chronic inflammation
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For instance, angiotensin-converting enzyme (ACE) inhibitor and angiotensin receptor blocker (ARB) drugs are used as an effective first-line therapy
