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The γ-glutamyl cycle, comprising six enzymes, harbors four hereditary defects: γ-glutamylcysteine synthetase, glutathione synthetase, γ-glutamyl transpeptidase, and 5-oxoprolinase

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In RBCs, NADPH are produced exclusively via HMP shunt by the action of G6PD and are responsible to maintain membrane integrity preventing premature destruction

We show that Cpt1a P479L mutant mice are resistant to rotenone-induced PD, and that inhibition of CPT1 is capable of restoring neurological function, normal glucose metabolism, and alleviate markers of PD in the midbrain