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When the equilibrium between the antioxidants and oxidants is compromised then it leads to oxidative stress

However, during a situation where massive apoptosis cannot be completed and converts into secondary necrosis with cell swelling and cell contents release, the extensive caspase activation of the primary apoptosis is still measurable [44]

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Overall, these results suggest that C15ORF48 is an inducer of starvation-independent macroautophagy, that activates AMPK-ULK1 signaling axis, which is triggered by reduction of intracellular ATP levels due to mild repression of mitochondrial activity