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19 It is also now clear that CYP-derived EETs also protect the heart against acute ischemia-reperfusion injury and chronic non-ischemic cardiomyopathy and hypertension

Additionally, the increased CHAC1 mRNA decay, GSH accumulation, ROS scavenging, drug resistant phenotypes in vitro and in vivo induced by GDIL overexpression was blocked when fragment 1 (1200 nt) in GDIL was deleted (Supplementary Fig