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Regarding these challenges, more efforts are needed to offer encouraging possibilities for the targeted and individualized use of biological rhythm therapy
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October 22, 2020
Knockdown studies of GPX4 results in a reduction in the expression of genes encoding components of Complex I, IV, and V [53], while overexpression of mitochondrial GPX4 prevents release of the proapoptotic molecule cytochrome C from mitochondria, suggesting a key role as an anti-apoptotic agent in mitochondrial death pathways [26]
Yeo SK, Ooi LG, Lim TJ, Liong MT
