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In vivo solution phase complement activation assays Homozygous human FcRn transgenic mice (B6.Cg-Fcgrttm1Dcr Tg(FcGRT)32Dcr/DcrJ) (Tg32) (The Jackson Laboratory) were injected i.v
Reduction by GSSG was significantly slower, but not absent, showing that GSH-deficient cells can use glutathione reductase to restore their highly reductive cellular glutathione redox state from the oxidant GSSG
Chou SM, Han CY, Wang HS et al (1999) A receptor for advanced glycosylation endproducts (AGEs) is colocalized with neurofilament-bound AGEs and SOD1 in motoneurons of ALS: immunohistochemical study
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