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Complementary clinical and meta-analytic data consistently demonstrate systemic redox imbalance, including lower glutathione reserves, altered GSH/GSSG ratios, increased lipid peroxidation, and dysregulated NRF2 signaling, linking oxidative status to immune activation and synaptic dysfunction (James et al., 2004

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doi: 10.1128/AEM.00867-17

Histology of the lesions revealed intense superficial edema with a moderate perivascular and interstitial inflammatory infiltrate formed of lymphocytes, histiocytes, and numerous eosinophils, occupying the full thickness of the dermis and infiltrating the subcutaneous adipose tissue

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