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Similar to NNMT overexpression, MNA-treated hepatocytes show a dose-dependent increase in glucose production, Sirt1 protein expression, G6pc mRNA expression, and Pck1 mRNA expression compared to controls and these changes caused by MNA treatment can be abolished by Sirt1 knockdown [14]

SLU-PP-332 is currently being studied for its interaction with metabolic pathways and its potential role in energy regulation and mitochondrial function