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Zheng J, Lu J, Mei S, Wu H, Sun Z, Fang Y, Xu S, Wang X, Shi L, Xu W, Chen S, Yu J, Liang F, Zhang J

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The presence of NiO NPs amplified the cytotoxic effects of aged -synuclein amyloid protein by promoting the production of high levels of ROS, inactivating SOD and catalase, and inducing inflammation, as evidenced by elevated levels of tumor necrosis factor-alpha (TNF-), IL-1, and IL-1

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Baseline MELD-3 AUC = 0.650) suggests that disease trajectory during treatment, rather than treatment itself, is the dominant factor determining outcomes