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Excessive release of glutamate and its binding with NMDA, -amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA), or kainite (KA) receptors promotes excitotoxicity cell injury with a consequent activation of many molecular pathways, resulting in intracellular calcium (Ca 2+ ) overload 90,91 , reactive oxygen species (ROS) production, mPT, and cell swelling with brain edema 92

This resulted in normalization of the tGSH:GSSG ratio, also known as the glutathione redox profile

Safety and efficacy of a Na v 1.7 selective sodium channel blocker in patients with trigeminal neuralgia: a double-blind, placebo-controlled, randomised withdrawal phase 2a trial

Atpenin A5 is a known SDHB inhibitor 52 and was previously assessed for its therapeutic effect on cardiac injury in isolated perfused rat hearts 53