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Schnakers C, Lutkenhoff ES, Bio BJ, McArthur DL, Vespa PM, Monti MM
Inhibition of p38-MAPK potentiates cisplatin-induced apoptosis via GSH depletion and increases intracellular drug accumulation in growth-arrested kidney tubular epithelial cells
These protective effects were attributed to antioxidant mechanisms that mitigated oxidative stress, as well as the inhibition of key inflammatory mediators, including TGF-, TNF-, and p53
Additionally, because superoxide does not easily cross membranes at a neutral pH, E
The effect of nanoparticle size, shape, and surface chemistry on biological systems
