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Hence, either the chronic AA overload in flu-1 worms might impair LRO function or perhaps flu-1 might be partially involved in BA-induced autofluorescence and in the adaptive response to BA toxicity
In these cases, modestly increasing intake can help maintain stable antioxidant capacity
Inhibition of IDH1 also sensitized CCNE1-driven tumours to olaparib in vivo (Fig
Pharmacoeconomics 2008
