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Even if antioxidant defense is significantly increased and oxidative stress markers are reduced, antioxidant treatment may have no significant impact on disease progression [161]
Therefore, DHODH has emerged as a potential regulatory hub, where its inhibition may enhance ferroptotic cell death, offering a novel angle for therapeutic intervention, particularly in cancers such as pancreatic adenocarcinoma (175) and breast cancer (176), where certain cells show resistance to other forms of cell death (171,177)
Regarding corticosterone levels, which directly indicate stress in murine models, a decrease was noted shortly after DSIP exposure
doi: 10.1186/2193-1801-3-578
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