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Subsequent study showed that GCLC is phosphorylated directly by activation of protein kinase A (PKA), protein kinase C (PKC) or Ca 2+ -calmodulin kinase II (CMK) (Sun et al., 1996)
[DOI] [PMC free article] [PubMed] [Google Scholar] 99.Jiao Y., Yu Y., Li B., Gu X., Xie K., Wang G., Yu Y

Additionally, MJT promotes ferroptosis and alleviates iron dysregulation and LPO during atherogenesis, reducing DMT1 and SOCS1/p53 expression through STAT1 phosphorylation (Shi et al., 2024)
The neuropathology of neurodegeneration with brain iron accumulation

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