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By inhibiting atrial-specific potassium currents and decreasing Na+ -Ca2+ exchange current, it reduces delayed after depolarizations and arrhythmia risk

*Correspondence: Luca Cucullo, [email protected] This article was submitted to Neuropharmacology, a section of the journal Frontiers in Pharmacology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers