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Mechanistically, gut microbiota-derived SCFAs exert their functions by activating trans-membrane G-protein-coupled receptors (GPCRs) of GPR41 and GPR43 or inhibiting histone acetylation (HDAC) to directly enter host cells (Moniri and Farah, 2021)

These predicted targets were further integrated with additional experimental information on the relevance of enzymes for viral replication to subselect candidates for experimental testing

doi: 10.1016/j.jtemb.2017.06.004

However, little is known about how this fusion process is regulated and controlled during APAP-induced liver injury

doi: 10.7150/thno.97005 79 ZhuangHLvQZhongCCuiYHeLZhangCet al