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Through laboratoryexperimentationon various animals, including rodents, pigs, dogs, and humans, the unique mechanism of AOD-9604 has been identified, which primarily stimulates the release of fat from adipose tissue, inhibits the accumulation of new fat in adipocytes, and promotes fat oxidation

nerve damage).It showed that after8 weeks of treatment, patients treated with mecobalamin injections showed signs of nerves being repaired, while no effects were seen in those taking the tablets. Some background Diabetic peripheral neuropathy(DPN)is []

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