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Capmatinib and Tepotinib are designed as a selective small-molecule inhibitors that indirectly prevent the activation of downstream signaling by blocking c-MET phosphorylation at the start, by binding to the ATP-binding pocket of the kinase in its active conformation [112]
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10.1038/s41467-020-20313-x [DOI] [PMC free article] [PubMed] [Google Scholar]
The cofactor required to form methyl conjugates is adenosylmethionine (SAM), which is primarily formed by the condensation of ATP and Lmethionine
Maeda, A
