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To determine whether Zfp423 plays a role in Dmrta2 repressive function, we first examine the consequences of its overexpression on the ability of Dmrta2 to repress the E60 enhancer in P19 cells

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Loss- or gain-of-function studies of Notch/RBPjk signaling showed that chondrogenesis was either enhanced or impaired, respectively, in mouse embryos

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