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Mutagenicity of 5-aza-2-deoxycytidine is mediated by the mammalian DNA methyltransferase
doi: 10.1001/amajethics.2019.347, 179
These encompass the activation of iron-regulatory proteins, thereby elevating transferrin receptor expression, and the repression of the transcription factor CCAAT/enhancer-binding protein (C/EBP) or bone morphogenetic protein (BMP)-mediated Smad signaling pathways, resulting in attenuated hepcidin expression [779781]
Clark PM, Holder RL, Haque SM, Hobbs FD, Roberts LM, Franklyn JA
