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Radiotherapy resistant HIF1 KO NSCLC cells further induce ser/gly synthesis pathway following irradiation, a response non-existent in HIF2 KO cells The observations of a distinct metabolic reprogramming and redox defense mechanism of NCI-H1299 NSCLC cells upon loss of HIF1 and HIF2 might imply divergent mechanisms for their survival in response to oxygen-dependent treatments such as radiotherapy

[DOI] [PubMed] [Google Scholar] Estonius M, Svensson S, et al

Once the drip begins, you can read, listen to music, or just relax

doi: 10.1146/annurev.psych.48.1.191

A study indicated that glutathione can improve insulin sensitivity, helping cells use glucose more effectively