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Similarly, in LDL receptor knock-out mice subjected to a Western diet, treatment-reduced plaque lesions and semaglutide were reported to affect the expression of several genes related to cholesterol metabolism, leukocyte recruitment, adhesion and extravasation, and also extracellular matrix protein turnover [35]

A recent study in Caenorhabditis elegans revealed that early-life B12 deficiency caused increase in the levels of fat and demonstrated that the methionine cycle-SBP- 1/SREBP1-lipogenesis axis was required for early-life B12 to determine health outcomes later in life [30]

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All participants provided written informed consent for brain donation upon death

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