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While the mechanisms underlying APAP-mediated lysosomal instability or permeabilization are not fully understood, the iron chelator desferal prevented mitochondrial depolarization and hepatocellular death after APAP exposure (Kon et al., 2010)

We found that ALCAR was able to limit the production of CXCR4 and CXCL12 in PCa (PC-3, LNCaP, DU145) and BPH cell lines, suggesting a potential role in limiting the induction of a migratory phenotype (Fig

*Correspondence: Vishwanath Venketaraman, [email protected] This article was submitted to Clinical Microbiology, a section of the journal Frontiers in Cellular and Infection Microbiology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers