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Several studies have reported pathological astrogliosis and associated neuronal hypotrophic glucose metabolism in patients with neurodegenerative disease and diabetic model animals, showing increased glial fibrillary acidic protein (GFAP) and significant cellular hypertrophy (32), which has been correlated to some extent with the severity of cognitive impairment in patients with Alzheimers disease (AD)

This is accompanied by the upregulation of pathways that mediate antigen presentation [e.g., major histocompatibility complex (MHC) II], phagocytosis related pathways such as C3, C4 and FCGR4, CD86 (Lagraoui et al., 2012), pathways that promote pro-inflammatory cytokines (e.g., IL-10, IL-6, IL-2, IFN-, IL-1) and chemokines expression (e.g., CXCL4, CCL4, CCL2)

6 Within one year of a patients initial episode, those with HZO have a risk of stroke 4.3 times higher than that of all HZ patients (whose risk is 1.3 times that of controls)

A novel dodecadepsipeptide, cereulide, isolated from Bacillus cereus causes vacuole formation in HEp-2 cells