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To determine whether the death of glioma cells induced by C5aR1 knockdown is due primarily to ferroptosis, we performed TEM, which showed that C5aR1 knockdown caused extensive changes in mitochondria, such as mitochondrial shrinkage, loss of cristae, and rupture of the outer membrane (Fig

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Recent integrative evidence from expands on canonical molecular pathways by illustrating how vitamin D and physical exercise converge on shared neurotrophic and anti-inflammatory signaling networks relevant to neuroplasticity and muscle metabolism