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Stasi R, Abriani L, Beccaglia P, Terzoli E, Amadori S

Our previous data also suggests that maternal SE impairs brain mitochondrial levels of oxidative phosphorylation (OXPHOS) complexes in male offspring with reduced endogenous antioxidant capacity from birth (Chan et al., Furthermore, as SE offspring have reduced brain antioxidants and increased cellular oxidative damage (Chan et al., 2015) and can provide neuroprotection by elevating brain antioxidant capacity in aging rats (Juliet et al., Materials and Methods Animals The animal experiments were approved by the Animal Care and Ethics Committee at the University of Technology Sydney (ACEC#2011-313A)

The Review on Antimicrobial Resistance

J Biol Chem 247: 832841, 1972

Effects of long-term vitamin E supplementation on cardiovascular events and cancer: a randomized controlled trial