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Sample Type(s): Mouse Embryo Primary Cells (ESCs), Abstract Full Text Oxidation resistance 1 prevents genome instability through maintenance of G2/M arrest in gamma-ray-irradiated cells: Matsui, A., Kobayashi, J., et al

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-hydroxybutyrate and its metabolic effects on age-associated pathology

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muI Na+ channels expressed transiently in human embryonic kidney cells: Biochemical and biophysical properties

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In cancer, ROS act as a double-edged sword: while some cancer therapies are based on triggering intracellular ROS to the point of cellular destruction 5 , ROS-derived genetic instability and protein oxidation can also work in favor of pro-oncogenic signaling by modulating metabolic pathways and supporting neoplastic transformation 5,6,7

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