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Taken together, these findings demonstrate that hepatocyte-specific FPN deficiency significantly promoted obesity and glucose intolerance during the development of diet-induced metabolic disorders
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4A) and confirmed histologically at the end-point, 38 days post-TAM injection (Supplementary Fig
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Increasing levels of cAMP activate PKA, which phosphorylates cAMP response element-binding protein (CREB) for further downstream gene expression regulation
