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10.1016/j.nmni.2021.100897 [DOI] [PMC free article] [PubMed] [Google Scholar] 324.Sestili P, Fimognari C
If there is any defect in DICER1, then most of these RNAs cannot be generated [107]
in fact, these treatments promoted UPR mt and mitophagy, restoring mitochondrial and organismal function, and even stimulated the reduction of amyloid deposits in these models 114
These timeframes align with the protocols used in published peptide efficacy studies
