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These results support the conclusion that simultaneous inhibition of GSH and Trx metabolism pathways induces oxidative stress and clonogenic killing in HNSCCs and this strategy may be useful in sensitizing HNSCCs to EGFR inhibitors.

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Regarding all other medications, our review found no evidence to contravene their use in normal therapeutic doses to G6PD-deficient patients.There is a need for evidence-based global consensus regarding medication use in G6PD-deficient patients.

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