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Intravenous administration might be better in some cases

BACH1 directly modulates numerous critical inflammatory and repairrelated genes in musclederived macrophages following cardiotoxin injury, including IGF1, SLC40A1, IL6, IL10, GDF3, PPARG, DUSP1, and CEBPB, potentially elucidating the delayed muscle regeneration phenotype observed in BACH1 KO mice

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