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These N-chlorinated serum proteins can then bind aggregating proteins and prevent their precipitation, activate immune cells, and protect them from cytotoxic antigens

25 Therefore, elevated blood tyrosine levels in HT-1 are more likely due to a secondary inhibition of proximal steps in tyrosine degradation and not the deficiency of FAH

GPNA, L--glutamyl-p-nitroanilide
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11) containing full-length (bHEV ORF2 fl) and a 5'-truncated deletion (bHEV ORF2 5' tr) of HEV ORF2 (Pakistan strain) were constructed by standard homologous recombination in Sf-9 insect cells
