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For example, luteolin is effective in therapeutically induced cytotoxicity of cancer cells due to its suppression of cell survival pathways, such as phosphatidylinositol-3-kinase (PI3K)/Akt, NF-B, and X-linked inhibitor of apoptosis protein (XIAP), resulting in the stimulation of apoptosis pathways, including the induction of the p53 tumour suppressor [39]

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This metabolite can accumulate to high levels when -ECS is overexpressed alone, either in the cytosol or the chloroplast (Noctor et al., 1996, 1998a

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