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10.1073/pnas.96.13.7238 Proc

341Sanchez-ElsnerTRamirezJRSanz-RodriguezFVarelaEBernabeuCBotellaLM

Stockwell BR, Friedmann Angeli JPF, Bayir H, Bush AI, Conrad M, Dixon SJ, et al

Collectively, these findings indicate that non-selective or excessive chelation of brain iron may disrupt physiological metallostasis, emphasizing the need for precisely titrated, brain-targeted anti-ferroptotic interventions with balanced efficacytoxicity profiles. Beyond traditional chelators, several natural compounds have also shown potential in regulating iron transport proteins

Glutathione for detox then steps in during Phase II, where it conjugates with these modified toxins, attaching itself to them and transforming them into water-soluble compounds [5]