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doi: 10.1172/JCI113450 110 NorrelundHFrystykJJorgensenJOMollerNChristiansenJSOrskovHet al

Specifically, GSH levels returned to their control values, which agreed with the cell-free-based antioxidant activity assays in our previous results 48

Furthermore, host genetic polymorphisms of drug-metabolizing enzymes also account for the eradication failure of H

TDP-43 accumulation in inclusion body myopathy muscle suggests a common pathogenic mechanism with frontotemporal dementia

Consequently, the hypoxic adaptation and inflammatory responses induced by CIH may result in altered CB sensitivity, with HIF-1 potentially playing a pivotal role