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In animal studies, the offspring of vitamin D-deficient rodents show brain structural changes similar to those seen in schizophrenia, such as thinning of the cortex, enlargement of lateral ventricles, and increased brain size due to increased proliferation and reduced shedding of neurons (63)

Emerging results on genetic disease-modifiers show limited overlap with genetic loci involved in disease risk, highlighting the point that disease initiation and disease progression are not necessarily driven by the same mechanisms

In fact, even factors such as the capsule, which is a crucial virulence factor finely regulated, are associated with multiple ccs and can be present in carriage isolates (Oldfield et al., 2018

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