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Matsuda N, Lu H, Fukata Y, Noritake J, Gao H, Mukherjee S, Nemoto T, Fukata M, Poo MM (2009) Differential activity-dependent secretion of brain-derived neurotrophic factor from axon and dendrite

SIRT6 alleviates mitochondrial dysfunction, podocyte injury, and ferroptosis induced by HG through activating the Nrf2/GPX4 pathway and increasing SLC7A11, HO-1, and SOD2 expressions

Glutathione levels naturally decline with age and may also be reduced by stress, malnutrition, and environmental toxin exposure

Progressive, transgenerational changes in offspring phenotype and epigenotype following nutritional transition

Ferroptosis is also manifested in the antioxidant system, and it appears that the expression of glutathione peroxidase 4 (GPX4) decreases in the antioxidant system (lipid peroxide) [16-21]