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ChIP-seq analysis revealed that PHF3-GFP bound to expressed genes and that recruitment to chromatin was reduced upon deletion of the SPOC domain, indicating that the interaction with phosphorylated Pol II CTD is critical for PHF3 binding to chromatin (Supplementary Fig

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To view a copy of this license, visit About this article Cite this article Li, H., Toh, P., Tan, J

News release: the FDA accepts the complete response for clinical holds of FG-2216 * /FG-4592 for the treatment of anemia