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Mutant -synuclein permeabilizes these vesicles causing leakage of the dopamine into the cytoplasm where it autooxidizes producing hydrogen peroxide, superoxide molecules, and toxic dopamine-quinone species creating oxidative stress conditions [122]

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This restoration is crucial for conversion of GSSG to GSH, a mechanism that aids in the host cell defense against ROS and supports cellular function and growth ( N -acetylcysteine enhanced GSH levels in A549 cells, however, the mechanism that underlies this increase in intracellular GSH, and whether this involved enhanced GRed enzymatic activity was not explored

& Mufson, E

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