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Similarly, SF pretreatment protected neural cells against A-fragment cytotoxicity and apoptosis in part through upregulation of Bcl-2 [169]
One interpretation is that brain iron elevation in AD could be a reactive, possibly protective sequestration mechanism (for instance, immobilizing iron in plaques or ferritin)
This is implicated in perturbing many cellular processes in both motor neurons and non-neuronal cells, including neuroinflammation (Bruijn et al., 2004
log 2 = 5.918 at 9 h) and was overall amongst the top-induced genes (Additional file 1)
Lavoie, L
